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dad-12 superfusion system with a 8-tube perfusion manifold  (ALA Scientific Instruments)

 
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    ALA Scientific Instruments dad-12 superfusion system with a 8-tube perfusion manifold
    Dad 12 Superfusion System With A 8 Tube Perfusion Manifold, supplied by ALA Scientific Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/dad-12+perfusion+system/dad+12+superfusion+system/pmc11891579__SC___016___D4SC06949H___s001-93-21-24
    Average 90 stars, based on 1 article reviews
    dad-12 superfusion system with a 8-tube perfusion manifold - by Bioz Stars, 2026-10
    90/100 stars

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    other:

    Article Title: Conversion of the Ion Selectivity of the 5-HT3AReceptor from Cationic to Anionic Reveals a Conserved Feature of the Ligand-gated Ion Channel Superfamily
    Article Snippet: Drugs were applied in one of three ways: via a U-tube device capable of complete solution exchange within 100 ms (19), via a DAD-12 perfusion system (ALA Scientific Instruments Inc.) (21), or, for voltage ramp experiments, via a Warner Instruments SF-77B fast solution exchange system (22), both of which are capable of solution exchange in ;30 ms. Current-voltage relationships were established by measuring the peak current evoked by 10 mM 5-HT at holding potentials between 280 and 140 mV or by the application of voltage ramps (270 to 1100 mV, in 200 ms) during the plateau phase of an agonist-evoked response.

    Article Title: Evidence that brain-derived neurotrophic factor neuroprotection is linked to its ability to reverse the NMDA-induced inactivation of protein kinase C in cortical neurons.
    Article Snippet: Rapid NMDA application was accomplished using a DAD-12 perfusion system (ALA Scientific Instruments, Westbury, NY, U.S.A.).

    Article Title: An alternative Ca2+-dependent mechanism of neuroprotection by the metalloporphyrin class of superoxide dismutase mimetics.
    Article Snippet: This study challenges the conventional view that metalloporphyrins protect cultured cortical neurons in models of cerebral ischemia by acting as intracellular catalytic antioxidants [superoxide dismutase (SOD) mimetics].. High SOD-active Mnporphyrins meso-substituted with N,N′-dimethylimidazolium or N-alkylpyridinium groups did not protect neurons against oxygen-glucose deprivation (OGD), although lower SOD-active and -inactive para isomers protected against N-methyl-D-aspartate (NMDA) exposure.. Mnmeso-tetrakis(4-benzoic acid)porphyrin (MnTBAP), as well as SOD-inactive metalloTBAPs and other phenyl ringor β-substituted metalloporphyrins that contained redox-insensitive metals, protected cultures against OGD and NMDA neurotoxicity.



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